Stress Response

The stress response occupies a pivotal position in the depth-psychology corpus, functioning simultaneously as a physiological mechanism, a developmental variable, and a clinical target. The literature traces the concept from Hans Selye’s foundational General Adaptation Syndrome and Walter Cannon’s unitary organismic response through contemporary neuroscientific elaborations that distinguish between acute, adaptive activation and chronic, pathogenic dysregulation. Panksepp maps the pituitary-adrenal and sympathoadrenal axes with neurochemical precision, while Porges reframes the entire construct within Polyvagal Theory, arguing that stress is best understood not as an externally defined event but as a measurable disruption of homeostatic function — a reconceptualization that shifts clinical attention toward cues of safety rather than mere removal of threat. Lanius and colleagues situate the stress response within developmental traumatology, documenting how early life adversity sensitizes HPA axis functioning in ways that persist into adulthood, altering cortisol pulsatility, autonomic tone, and immunological resilience. Payne and Levine attend to the body’s capacity to complete and reverse stress-response cycles, foregrounding the somatic work of trauma therapy. Easwaran bridges psychophysiology and contemplative traditions, insisting that every habitual mental state carries a somatic stress signature. Across these positions, a central tension persists: whether the stress response is a discrete, correctable mechanism or an enduring organismic orientation shaped by relational history.

In the library

Hans Selye, MD, PhD, first described the stress response in the 1950s. He introduced the General Adaptation Syndrome model describing how the body responds to external sources of stress and how psychological stress influences physical illness.

This passage establishes Selye’s General Adaptation Syndrome as the foundational model of the stress response, linking hormonal dysregulation to disease and positing the body’s adaptive energy as a finite resource depleted by chronic stress.

Laurence Heller, Ph D, Healing Developmental Trauma How Early Trauma Affectsthesis

Dig deeper with Sebastian →

a sustained stress response can kill certain brain cells! At present, we know that this neurotoxic effect can be produced in both experimental laboratory animals and those confronting real-life stressors in the wild.

Panksepp demonstrates that the pituitary-adrenal and sympathoadrenal stress response systems, when chronically activated, produce cortisol-mediated neurotoxicity with lasting consequences for cognitive function.

Panksepp, Jaak, Affective Neuroscience The Foundations of Human and Animal, 1998thesis

Dig deeper with Sebastian →

A Polyvagal perspective shifts the discussion from the external features defining stress and threat to the nervous system’s ability to support or disrupt homeostatic functions (i.e., processes supporting health, growth, and restoration).

Porges reconceptualizes the stress response as a measurable disruption of homeostatic function rather than a reaction to external threat, repositioning the nervous system’s internal state as the primary clinical variable.

Porges, Stephen W., Polyvagal Theory: A Science of Safety, 2022thesis

Dig deeper with Sebastian →

Walter Cannon’s concept of the ‘stress response’ (Cannon, 1970), a supposedly unitary response of the organism to any stressor regardless of its nature. This early approach led to several difficulties.

Payne critically examines the historical assumption of a unitary stress response across biological and psychosocial stressors, tracing terminological and conceptual ambiguities back to Selye and Cannon.

Payne, Peter, Somatic experiencing: using interoception and proprioception as core elements of trauma therapy, 2015thesis

Dig deeper with Sebastian →

Stress is defined as a physically, mentally or emotionally disruptive or threatening condition occurring in response to adverse intrinsic or extrinsic influences, for which adequate coping resources are unavailable.

Lanius and colleagues anchor the stress response within developmental traumatology, emphasizing the role of inadequate coping resources and disrupted homeostasis in producing neuroendocrine vulnerability.

Lanius, edited by Ruth A, The impact of early life trauma on health and disease the, 2010thesis

Dig deeper with Sebastian →

Short disruptions or acute stress followed by rapid recoveries would function as neural exercises promoting resilience. While more chronic disruptions without periods of recovery would lead to disease and tissue/organ damage.

Porges distinguishes acute from chronic stress responses, arguing that recovery cycles build neural resilience while persistent disruption of homeostasis produces pathological sequelae.

Porges, Stephen W., Polyvagal Theory: A Science of Safety, 2022thesis

Dig deeper with Sebastian →

Levine’s approach suggests that to be ‘stuck’ in a ‘stressed-out’ or traumatized state is for the CRN to be stuck in a dysfunctional dynamic mode which is, in principle, fully reversible.

Within Somatic Experiencing, traumatic stress is understood as a reversible dysregulation of the central regulatory network rather than a fixed structural deficit, foregrounding the therapeutic potential of somatic intervention.

Payne, Peter, Somatic experiencing: using interoception and proprioception as core elements of trauma therapy, 2015thesis

Dig deeper with Sebastian →

the stress response, a general physiological arousal that activates defense mechanisms all over the body. Blood pressure is one of these defense mechanisms, and it is controlled by several factors sensitive to emotional states.

Easwaran integrates psychophysiological and contemplative perspectives, arguing that chronic habitual mental states generate a systemic stress response — including autonomic and endocrine effects — that compounds physical illness.

Easwaran, Eknath, The Upanishadssupporting

Dig deeper with Sebastian →

every chronic or habitual mental state includes effects on health. Often these effects include the stress response, a general physiological arousal that activates defense mechanisms all over the body.

From a contemplative standpoint, this passage argues that the stress response is not an isolated episode but the somatic expression of habitual mental patterns, linking psychological and physical disease.

Easwaran, Eknath, Essence of the Upanishads: A Key to Indian Spiritualitysupporting

Dig deeper with Sebastian →

The physiological state is an intervening variable that may buffer or exacerbate the effective impact of stress and trauma. Physiological adaptations and autonomic dysregulation are central to the PTSD diagnosis.

Haeyen applies Polyvagal Theory to creative arts therapies, framing the stress response as modulated by physiological state and neuroception, with clinical implications for shifting from defensive to prosocial autonomic modes.

Haeyen, Suzanne, A theoretical exploration of polyvagal theory in creative arts and psychomotor therapies for emotion regulation in stress and trauma, 2024supporting

Dig deeper with Sebastian →

maternal deprivation for 24 hours in 12-day-old rats produce heightened basal and stress-induced ACTH and altered CRFR expression in brain regions involved in the pathophysiology of depression and anxiety.

Animal model data demonstrate that early maternal deprivation produces enduring sensitization of the HPA-axis stress response, establishing a neurobiological pathway from early neglect to adult affective disorder.

Lanius, edited by Ruth A, The impact of early life trauma on health and disease the, 2010supporting

Dig deeper with Sebastian →

hormonal responses modulate the functioning of the HPA axis in ways that, if continued, may increase the risk of immune disorders and heighten sensitivity to future stress, potentially leading to cognitive deficits and social–emotional problems.

This passage establishes a cascade model in which early stress-response activation alters HPA functioning with downstream consequences for immunity, cognition, and social-emotional development.

Lanius, edited by Ruth A, The impact of early life trauma on health and disease the, 2010supporting

Dig deeper with Sebastian →

the prefrontal cortex plays an important role in mediating subjective sense of control, which, in turn, appears to be a critical feature of resilience to stress.

The passage links prefrontal regulation of perceived control to differential stress-response intensity, positioning cortical mediation as a key factor in resilience versus vulnerability.

Lanius, edited by Ruth A, The impact of early life trauma on health and disease the, 2010supporting

Dig deeper with Sebastian →

Women with abuse-related PTSD had decreased baseline cortisol based on 24 hour diurnal assessments of plasma, increased pulsatility of cortisol, and exaggerated cortisol response to stressors.

Clinical neuroendocrine data on PTSD document the long-term alteration of the cortisol stress response following childhood sexual abuse, linking HPA dysregulation to symptom severity.

Lanius, edited by Ruth A, The impact of early life trauma on health and disease the, 2010supporting

Dig deeper with Sebastian →

various forms of stress—drugs that effect monoamine activity, oxidative stress and a sudden elevation of temperature—induce a universal common pathway of transcription and translocation of particular genes that are not expressed under normal physiological conditions.

Schore extends the stress-response concept to the cellular level, identifying stress-protein induction as a universal genomic pathway that during postnatal development serves both protective and potentially disruptive functions.

Schore, Allan N., Affect Regulation and the Origin of the Self: The Neurobiology of Emotional Development, 1994supporting

Dig deeper with Sebastian →

oxytocin can stimulate affiliative responses even in the face of threat by lowering blood pressure and heart rate. Partly, this system evolved to facilitate maternal behavior following the extreme stress of childbirth.

Fogel identifies the oxytocin-parasympathetic system as a counter-regulatory mechanism that can attenuate the stress response through affiliation, with implications for social buffering of trauma.

Fogel, Alan, Body Sense: The Science and Practice of Embodied Self-Awareness, 2009supporting

Dig deeper with Sebastian →

low social support has been shown to be associated with exaggerated cardiovascular and neuroendocrine responses to laboratory stressors.

The passage documents social support as a significant moderator of the stress response, with isolation amplifying both cardiovascular and HPA reactivity in both animal and human studies.

Lanius, edited by Ruth A, The impact of early life trauma on health and disease the, 2010supporting

Dig deeper with Sebastian →

The stress response involves a complex cascade of neural and endocrine pathways in response to a potentially harmful stimulus, altering the body state and subsequently interoceptive perception.

Lovelock establishes that the stress response modifies interoceptive perception, thereby influencing subjective sensitivity to pharmacological agents such as alcohol and generating vulnerability to addictive patterns.

Lovelock, Dennis F., Interoception and alcohol: Mechanisms, networks, and implications, 2021supporting

Dig deeper with Sebastian →

anger, toward the self or others, is one of several feelings that trigger the release of stress hormones into our bloodstreams. These hormones cause our coronary arteries to constrict.

Dayton connects specific emotional states, particularly anger, to discrete cardiovascular components of the stress response, illustrating how unprocessed relational emotion becomes a physiological health risk.

Dayton, Tian, Emotional Sobriety: From Relationship Trauma to Resilience and Lasting Fulfillment, 2007supporting

Dig deeper with Sebastian →

much of this research was conducted by psychodynamically oriented researchers who tended to emphasize the individual’s predisposition to the stress response.

Pargament situates early psychodynamic research within the history of stress-response studies, noting the field’s shift from individual predisposition toward generalized patterns of crisis reaction.

Pargament, Kenneth I, The psychology of religion and coping theory, research,, 2001supporting

Dig deeper with Sebastian →

a physiological state characterized by a vagal withdrawal would support the mobilization behaviors of fight and flight. In contrast, a physiological state characterized by increased vagal influence on the heart would support spontaneous social engagement behaviors.

Porges describes how distinct autonomic states underlie different behavioral repertoires, providing the neurophysiological substrate that determines the character and intensity of stress-response mobilization.

Porges, Stephen W., The Polyvagal Theory: Neurophysiological Foundations of Emotions, Attachment, Communication, and Self-Regulation, 2011aside

Dig deeper with Sebastian →

Related terms